Kids Disease Child Disease Encyclopedia
Illustration representing Impetigo Contagiosa
Mild Bacterial Pyodermas & Super-infections

Impetigo Contagiosa

Superficial Bacterial Epidermal Infection

Primary risk age: Preschool and school-aged children (2 to 5 years; highly contagious)

Urgency
Mild
Typical age
Preschool and school-aged children (2 to 5 years; highly contagious)
Body system
Dermatological System

Typical course: Topical antibiotic therapy typically resolves the infection within 5 to 7 days.

Reviewed against AAP · CDC · WHO · NHS guidance Last reviewed 2026-06-13

1. Summary & Pathophysiology

Superficial Bacterial Epidermal Infection

Pathophysiology (Development Path)

Bacteria enter the superficial epidermis through minor breaks in the skin (scratches, insect bites, eczema). The bacteria produce exfoliative toxins that break down cell adhesion molecules in the upper skin layers, causing small blisters that quickly rupture.

Primary Causes & Etiology

Staphylococcus aureus primarily; Streptococcus pyogenes (Group A Strep) secondary.

2. Symptom Continuum

  1. Early Onset Signs

    Small, red macules or papules appearing around the mouth, nose, or exposed extremities, which quickly turn into small vesicles.

  2. Progressive Phase

    The vesicles rupture, releasing a serous fluid that dries to form characteristic "honey-colored" crusts on a red, moist base. The lesions are itchy but generally painless.

  3. Severe Indicators

    Bullous Impetigo: marked by larger, flaccid, fluid-filled blisters (bullae) that persist and are accompanied by systemic signs like mild fever and diarrhea. Ecthyma: a deeper form of impetigo that penetrates into the dermis, causing painful, crusted ulcers that heal with scarring.

3. Clinical Verification

Clinical diagnosis based on the characteristic honey-colored crusts. Gram stain or culture of the wound fluid can confirm the bacterial source if the infection is resistant to therapy.

4. Care & Elements Plan

Primary Care Treatment Plan

Targeted topical or oral antibiotics depending on the extent of the lesions. Gently wash the area and remove crusts to allow topical antibiotics to penetrate.

Home Support Elements

Soak crusts with warm, soapy water to gently remove them. Apply prescribed topical ointments. Cut the child's fingernails short and keep them from scratching to prevent the infection from spreading (autoinoculation).

Generic Active Ingredients (No Brands)

  • Mupirocin (generic topical antibiotic active ingredient for localized lesions)
  • Cephalexin (generic oral antibiotic active ingredient for widespread or bullous infections).

Lists active elements only. Never administer self-designed therapies.

5. Doctor Critical Lines

Critical Thresholds: When to See a Doctor

Consult a doctor if the lesions do not improve after 48 hours of starting topical antibiotics, or if the child develops a fever or worsening redness around the sores.

6. Vaccine & Prevention

Routine Prophylaxis (Prevention)

Keep fingernails short and clean. Practice strict hand washing. Wash towels, bedding, and toys of an infected child separately. Keep cuts and scrapes clean and covered.

Immunization Context

No specific immunizations are associated with this bacterial skin infection.

7. Timelines & Outlook

Active Timeline

Topical antibiotic therapy typically resolves the infection within 5 to 7 days.

Expected Prognosis

Excellent. The infection resolves completely without scarring (except in ecthyma cases) with appropriate antibiotic therapy.

Potential Untreated Complications

Post-streptococcal glomerulonephritis (if caused by Streptococcus pyogenes), cellulitis, and scarring (in ecthyma).