Impetigo Contagiosa
Superficial Bacterial Epidermal Infection
Primary risk age: Preschool and school-aged children (2 to 5 years; highly contagious)
- Urgency
- Mild
- Typical age
- Preschool and school-aged children (2 to 5 years; highly contagious)
- Body system
- Dermatological System
Typical course: Topical antibiotic therapy typically resolves the infection within 5 to 7 days.
Reviewed against AAP · CDC · WHO · NHS guidance Last reviewed 2026-06-13
1. Summary & Pathophysiology
Superficial Bacterial Epidermal Infection
Pathophysiology (Development Path)
Bacteria enter the superficial epidermis through minor breaks in the skin (scratches, insect bites, eczema). The bacteria produce exfoliative toxins that break down cell adhesion molecules in the upper skin layers, causing small blisters that quickly rupture.
Primary Causes & Etiology
Staphylococcus aureus primarily; Streptococcus pyogenes (Group A Strep) secondary.
2. Symptom Continuum
- Early Onset Signs
Small, red macules or papules appearing around the mouth, nose, or exposed extremities, which quickly turn into small vesicles.
- Progressive Phase
The vesicles rupture, releasing a serous fluid that dries to form characteristic "honey-colored" crusts on a red, moist base. The lesions are itchy but generally painless.
- Severe Indicators
Bullous Impetigo: marked by larger, flaccid, fluid-filled blisters (bullae) that persist and are accompanied by systemic signs like mild fever and diarrhea. Ecthyma: a deeper form of impetigo that penetrates into the dermis, causing painful, crusted ulcers that heal with scarring.
3. Clinical Verification
Clinical diagnosis based on the characteristic honey-colored crusts. Gram stain or culture of the wound fluid can confirm the bacterial source if the infection is resistant to therapy.
4. Care & Elements Plan
Primary Care Treatment Plan
Targeted topical or oral antibiotics depending on the extent of the lesions. Gently wash the area and remove crusts to allow topical antibiotics to penetrate.
Home Support Elements
Soak crusts with warm, soapy water to gently remove them. Apply prescribed topical ointments. Cut the child's fingernails short and keep them from scratching to prevent the infection from spreading (autoinoculation).
Generic Active Ingredients (No Brands)
- Mupirocin (generic topical antibiotic active ingredient for localized lesions)
- Cephalexin (generic oral antibiotic active ingredient for widespread or bullous infections).
Lists active elements only. Never administer self-designed therapies.
5. Doctor Critical Lines
Critical Thresholds: When to See a Doctor
Consult a doctor if the lesions do not improve after 48 hours of starting topical antibiotics, or if the child develops a fever or worsening redness around the sores.
6. Vaccine & Prevention
Routine Prophylaxis (Prevention)
Keep fingernails short and clean. Practice strict hand washing. Wash towels, bedding, and toys of an infected child separately. Keep cuts and scrapes clean and covered.
Immunization Context
No specific immunizations are associated with this bacterial skin infection.
7. Timelines & Outlook
Active Timeline
Topical antibiotic therapy typically resolves the infection within 5 to 7 days.
Expected Prognosis
Excellent. The infection resolves completely without scarring (except in ecthyma cases) with appropriate antibiotic therapy.
Potential Untreated Complications
Post-streptococcal glomerulonephritis (if caused by Streptococcus pyogenes), cellulitis, and scarring (in ecthyma).
More in Bacterial Pyodermas & Super-infections
Hand, Foot, and Mouth Disease (HFMD)
Enteroviral Mucocutaneous Vesicular Eruption
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All ages; in infants often involves palms, soles, and the scalp.
Ringworm (Tinea)
Common contagious fungal skin infection (not a worm).
All ages; scalp ringworm is most common in school-age children.